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Number of items: 12.

Rothkamm, K. and Gunasekara, K. and Warda, S. and Krempler, A. and Löbrich, M. (2008):
Radiation-induced HPRT mutations resulting from misrejoined DNA double-strand breaks.
In: Radiation Research, pp. 639-648, 169, (6), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/18494542?dopt=Citation],

Kühne, M. and Riballo, E. and Rief, N. and Rothkamm, K. and Jeggo, P. A. and Löbrich, M. (2004):
A double-strand break repair defect in ATM-deficient cells contributes to radiosensitivity.
In: Cancer Research, pp. 500-508, 64, (2), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/14744762?dopt=Citation],

Krüger, I. and Rothkamm, K. and Löbrich, M. (2004):
Enhanced fidelity for rejoining radiation-induced DNA double-strand breaks in the G2 phase of Chinese hamster ovary cells.
In: Nucleic Acids Research, pp. 2677-2684, 32, (9), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/15148355?dopt=Citation],

Rothkamm, K. and Krüger, I. and Thompson, L. H. and Löbrich, M. (2003):
Pathways of DNA double-strand break repair during the mammalian cell cycle.
In: Molecular Cell Biology, pp. 5706-5715, 23, (16), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/12897142?dopt=Citation],

Rothkamm, K. and Löbrich, M. (2003):
Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses.
In: Proceedings of the National Academy of Sciences USA, pp. 5057-5062, 100, (9), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/12679524?dopt=Citation],

Rothkamm, K. and Löbrich, M. (2002):
Misrepair of radiation-induced DNA double-strand breaks and its relevance for tumorigenesis and cancer treatment (review).
In: International Journal of Oncology, pp. 433-440, 21, (2), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/12118342?dopt=Citation],

Kühne, M. and Rothkamm, K. and Löbrich, M. (2002):
Physical and biological parameters affecting DNA double strand break misrejoining in mammalian cells.
In: Radiation Protection Dosimetry, pp. 129-132, 99, (1-4), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/12194264?dopt=Citation],

Rothkamm, K. and Kühne, M. and Jeggo, P. A. and Löbrich, M. (2001):
Radiation-induced genomic rearrangements formed by nonhomologous end-joining of DNA double-strand breaks.
In: Cancer Research, pp. 3886-3893, 61, (10), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/11358801?dopt=Citation],

Tabocchini, M. A. and Rothkamm, K. and Signoretti, C. and Risse, J. and Sapora, O. and Löbrich, M. (2000):
Formation and repair of DNA double-strand breaks in gamma-irradiated K562 cells undergoing erythroid differentiation.
In: Mutation Research, pp. 71-82, 461, (1), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/10980413?dopt=Citation],

Kühne, M. and Rothkamm, K. and Löbrich, M. (2000):
No dose-dependence of DNA double-strand break misrejoining following alpha-particle irradiation.
In: International Journal of Radiation Biology, pp. 891-900, 76, (7), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/10923613?dopt=Citation],

Löbrich, M. and Kühne, M. and Wetzel, J. and Rothkamm, K. (2000):
Joining of correct and incorrect DNA double-strand break ends in normal human and ataxia telangiectasia fibroblasts.
In: Genes Chromosomes Cancer, pp. 59-68, 27, (1), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/10564587?dopt=Citation],

Rothkamm, K. and Löbrich, M. (1999):
Misrejoining of DNA double-strand breaks in primary and transformed human and rodent cells: a comparison between the HPRT region and other genomic locations.
In: Mutation Research, pp. 193-205, 433, (3), [Online-Edition: http://www.ncbi.nlm.nih.gov/pubmed/10343652?dopt=Citation],

This list was generated on Sat Oct 19 00:58:01 2019 CEST.