Theodoridis, Theodoros M. ; Tegos, Sotiris A. ; Diamantoulakis, Panagiotis D. ; Jamali, Vahid ; Karagiannidis, George K. (2023)
Glucose Regulation Through Cooperative Molecular Communication.
In: IEEE Communications Letters, 27 (11)
doi: 10.1109/LCOMM.2023.3315012
Artikel, Bibliographie
Kurzbeschreibung (Abstract)
In Type 1 diabetes, the pancreatic beta cells responsible for producing insulin are destroyed by the immune system. Insulin is needed to activate an insulin-dependent glucose transporter, which is responsible for taking glucose into the muscle cell for metabolism. Recent advances in nanotechnology, bioengineering and synthetic biology are bringing the artificial beta cell (ABC) closer to reality. In this letter, we model glucose regulation by ABCs as a cooperative molecular communication system, in which the glucose source is seen as the transmitter and the muscle as the receiver. the last absorbs the glucose in the presence of insulin, and the ABC is modeled as a a decode-and-forward relay that detects glucose molecules and releases insulin in response. Using this model, we analyze the end-to-end system performance for ABC-assisted glucose regulation by providing closed-form expressions for the probabilities of hyperglycemia and hypoglycemia and the error probability of the system. In addition, we present simulation results for quantifying performance and validation of the analysis.
| Typ des Eintrags: | Artikel |
|---|---|
| Erschienen: | 2023 |
| Autor(en): | Theodoridis, Theodoros M. ; Tegos, Sotiris A. ; Diamantoulakis, Panagiotis D. ; Jamali, Vahid ; Karagiannidis, George K. |
| Art des Eintrags: | Bibliographie |
| Titel: | Glucose Regulation Through Cooperative Molecular Communication |
| Sprache: | Englisch |
| Publikationsjahr: | November 2023 |
| Verlag: | IEEE |
| Titel der Zeitschrift, Zeitung oder Schriftenreihe: | IEEE Communications Letters |
| Jahrgang/Volume einer Zeitschrift: | 27 |
| (Heft-)Nummer: | 11 |
| DOI: | 10.1109/LCOMM.2023.3315012 |
| Kurzbeschreibung (Abstract): | In Type 1 diabetes, the pancreatic beta cells responsible for producing insulin are destroyed by the immune system. Insulin is needed to activate an insulin-dependent glucose transporter, which is responsible for taking glucose into the muscle cell for metabolism. Recent advances in nanotechnology, bioengineering and synthetic biology are bringing the artificial beta cell (ABC) closer to reality. In this letter, we model glucose regulation by ABCs as a cooperative molecular communication system, in which the glucose source is seen as the transmitter and the muscle as the receiver. the last absorbs the glucose in the presence of insulin, and the ABC is modeled as a a decode-and-forward relay that detects glucose molecules and releases insulin in response. Using this model, we analyze the end-to-end system performance for ABC-assisted glucose regulation by providing closed-form expressions for the probabilities of hyperglycemia and hypoglycemia and the error probability of the system. In addition, we present simulation results for quantifying performance and validation of the analysis. |
| Freie Schlagworte: | emergenCITY, emergenCITY_KOM |
| Fachbereich(e)/-gebiet(e): | 18 Fachbereich Elektrotechnik und Informationstechnik 18 Fachbereich Elektrotechnik und Informationstechnik > Resiliente Kommunikationssysteme (RCS) LOEWE LOEWE > LOEWE-Zentren LOEWE > LOEWE-Zentren > emergenCITY |
| Hinterlegungsdatum: | 20 Jun 2024 13:41 |
| Letzte Änderung: | 20 Nov 2024 12:45 |
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